Fresh Baked: The immediate metabolic effects of grain consumption

There are numerous long-term health repercussions of wheat and grain intake. The list of long-lasting effects of wheat/grain consumption includes initiation of autoimmune illness such as rheumatoid arthritis and type 1 diabetes, weight gain and weight problems, type 2 diabetes, oral decay, wear and tear of joint cartilage and osteoarthritis, and cataracts.
There are also short-term, or immediate, repercussions of wheat and grain intake, impacts that need only minutes to hours to develop, some results even developing within seconds. Such immediate effects include:

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Rise in blood insulin– The rise in blood sugar activates an increase in insulin. A desirable fasting insulin level is 4 mIU/L or less; people with insulin resistance have fasting insulin levels of 30,60, or 100 mIU/L. If anything consisting of wheat germ agglutinin (wheat, rye, barley, rice) crosses your lips, CCK is obstructed and the gallbladder does not get the full-strength signal to agreement. Changes in bowel flora– Improvements observed in microbiome composition when wheat and grains are removed suggest that wheat/grain consumption is associated with lowered species diversity and reductions in important species such as Akkermansia and Collinsella. Minimized absorption of minerals– The phytates of wheat and grains avidly bind minerals in the gut, obstructing their absorption.

I hope you appreciate that these are not benign or meaningless procedures. The immediate effects of taking in wheat and grains are profound.
The development of insulin resistance, for example, that over time leads to type 2 diabetes, infertility in girls with PCOS, or fatty liver, yields no outward symptoms in early phases. It is not unusual, for instance, to feel normal while insulin resistance rages beneath the surface up until you experience a heart attack.
What makes this all the more remarkable, obviously, is that wheat and grains are the beloveds of dietitians, medical professionals, and national dietary guidelines, yet they are debilitating to both long-term and near-term health. If all of this is new to you, you can catch up by starting with the Wheat Belly Revised & & Expanded edition of the book that brought all these concerns to light, upgraded with all new Wheat Belly techniques.

Rise in blood sugar– The amylopectin A carbohydrate of wheat and grains is rapidly broken down into glucose that raises blood sugar substantially. A blood sugar of 150-200 mg/dl prevails after, say, a bagel or bowl of (unsweetened) breakfast cereal, greater if there is any degree of insulin resistance present.
Increase in blood insulin– The increase in blood sugar level sets off a rise in insulin. If knowledgeable consistently, this leads to insulin resistance, i. e, bad responsiveness of muscle, liver, brain, and other organs to insulin that causes the pancreas to increase its production of insulin 10- to 100-fold. A preferable fasting insulin level is 4 mIU/L or less; individuals with insulin resistance have fasting insulin levels of 30,60, or 100 mIU/L. Recall that insulin triggers weight gain and obstructs weight-loss.
VLDL particles have the capability to gain entry into the walls of arteries, contributing to atherosclerosis that, over time, leads to occasions like heart attack. Lipogenesis is amplified in the existence of insulin resistance.
Development of small LDL particles– VLDL particles communicate in the blood stream with LDL particles, enriching them in triglycerides. Triglyceride-enriched LDL particles then go through a series of enzymatic “remodeling” reactions that result in development of small LDL particles that are powerful contributors to heart disease, especially considering that they continue in the bloodstream 5-7 times longer than more benign large LDL particles.
Triglyceride deposition in the liver– For uncertain reasons, some triglycerides resulting from lipogenesis stay in the liver and accumulate in time. Combined with swelling (such as that arising from bacterial endotoxemia from dysbiosis), this procedure results in fatty liver.
Stopping of cholecystokinin (CCK)– CCK is the hormone accountable for triggering the gallbladder to agreement to launch bile into the duodenum, thus starting the procedure of fat food digestion. If anything including wheat bacterium agglutinin (wheat, rye, barley, rice) crosses your lips, CCK is obstructed and the gallbladder does not receive the full-strength signal to agreement. With time, this causes bile tension that, in turn, results in crystal formation and gallstones.
Modifications in bowel plants– Improvements observed in microbiome structure when wheat and grains are gotten rid of recommend that wheat/grain usage is related to minimized species diversity and reductions in essential types such as Akkermansia and Collinsella. Modifications in bowel plants are surprisingly rapid. A boost in carbohydrate and sugar usage, for instance, triggers some species to bloom within hours.
Decreased absorption of minerals– The phytates of wheat and grains avidly bind minerals in the gut, blocking their absorption. This significantly reduces absorption of magnesium, manganese, zinc, iron, and calcium, each with their own ramifications for health. Iron deficiency anemia can be a particularly vexing issue, resulting in tiredness, shortness of breath, feeling cold, not to discuss medical treatments such as bone marrow biopsy and iron injections.

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